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Ann Clin Neurophysiol > Volume 15(2); 2013 > Article
Ann Clin Neurophysiol. 2013; 15(2): 63-67.
Published online December 31, 2013.
doi: https://doi.org/10.14253/kjcn.2013.15.2.63
Multiple Cranial Neuropathy and Polyradiculoneuropathy as Delayed Sequelae of Ethylene Glycol Intoxication
Min-Soo Kim1, Sun-Young Kim1, Jee-Hyun Kwon1, Wook-Joo Kim1, and Hyun-Chul Jung2
1Department of Neurology, Ulsan University Hospital, Ulsan University College of Medicine, Ulsan, Korea
2Department of Internal Medicine, Ulsan University Hospital, Ulsan University College of Medicine, Ulsan, Korea
Corresponding Author: Sun-Young Kim ,Tel: +82-52-250-8860, Fax: +82-52-250-7089, Email: biggirl77@medimail.co.kr
Received May 3, 2013    Accepted October 24, 2013
Copyright © 2013 The Korean Society of Clinical Neurophysiology
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
ABSTRACT
Multiple cranial and peripheral neuropathies as a delayed sequellae of ethylene glycol poisoning is a less well known clinical entity and its information about long-term electrophysiological and clinical outcomes is limited. We report a 45-year-old male who presented with acute renal failure and subsequently developed multiple cranial neuropathy, respiratory failure, and flaccid tetraparesis. Through sequential electrophysiological studies, we would like suggest that the main pathophysiology of ethylene glycol-related neuropathy is a demyelinating polyradiculoneuropathy with secondary axonal degeneration.
Key words: Ethylene glycol, Multiple cranial neuropathy, Polyneuropathy
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